@article{oai:mdu.repo.nii.ac.jp:00000740,
 author = {HATTORI, TOSHIMI and MAEHASHI, HIROSHI},
 issue = {1},
 journal = {松本歯学},
 month = {Apr},
 note = {application/pdf, The present study was conducted to determine whether stannic chloride (SnCl_4) at concentrations above 50цM inhibits or facilitates neuromuscular transmission. SnCl_4 decreased the amplitude of the endplate potential (e.p.p.) concentration-dependently in a concentration range from 0.05 to 0.5mM. Not only SnCl_4 (0.5mM) but also tartaric acid (TA, 0.5mM), a solvent for 0.5 mM SnCl_4, decreased the e.p.p.. The action of SnCl_4 was stronger than that of TA. Both SnCl_4 (0.5mM) and TA (0.5mM) decreased the quantal content of the e.p.p., and the action of the former was significantly stronger than that of the latter. SnCl_4 (0.5mM) raised the frequency of the miniature endplate potential (m.e.p.p.), whereas TA (0.5mM) reduced it. SnCl_4 (0.5mM) decreased the m.e.p.p. amplitude as did TA (0.5mM). The results obtained show that SnCl_4 inhibits neuromuscular transmission by decreasing the volume of the transmitters released by nerve impulses.},
 pages = {46--51},
 title = {Inhibition of Mouse Neuromuscular Transmission by Stannic Chloride},
 volume = {24},
 year = {1998}
}